By Tetsuzo Akutsu, Hitoshi Koyanagi, Setsuo Takatani, Kazunori Kataoka, Jack G. Copeland, Stuart L. Cooper, Peer M. Portner, David B. Geselowitz
Read or Download Artificial Heart 2: Proceedings of the 2nd International Symposium on Artificial Heart and Assist Device, August 13–14, 1987, Tokyo, Japan PDF
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Extra resources for Artificial Heart 2: Proceedings of the 2nd International Symposium on Artificial Heart and Assist Device, August 13–14, 1987, Tokyo, Japan
5 shows the variation of E' for the PCL(2000)EDA with time during the fatigue process. The sample for fatigue testing was immersed in a lipid solution for 30 and 96 days. TheE' of PCL(2000)EDA in the lipid solution is lower than that in air. This is attributed to the plasticization effect of lipids or water. The fatigue lifetime of SPUU decreased with an increase in lipid solution immersion time. This is a similar trend to that observed in air. These results may indicate that absorption of lipids under cyclic straining did not induce the reduction in fatigue strength.
7 [9-11). Polyurethanes carrying carboxylic acid ester groups were synthesized by the reaction of PTMG, MDI, and L-Lys-OMe, as shown at the top of Fig. 8. The surface of the polyurethane film was hydrolyzed and neutralized with citric acid. A polyurethane film on which carboxylic acid groups were introduced was synthesized, as shown at the bottom of Fig. 8. Heparin was coupled to the carboxylic acid groups of the polymer using 1-ethyl-3-(3-dimethylaminopropyl) carbodiimide, which is a water-soluble carbodiimide (WSC).
Biomaterials 7: 386-392 Discussion Kim (University of Utah): One of the methods you use to attach heparin to the surface is ionic bonding. How does the ionic exchange then operate? Ito (Kyoto University): Sodium heparinate was used. With ionically heparinized polyurethane, heparin is released. But in the initial stages of contact between polyurethane and blood, if the initial antithrombogenic surface is completed it is not necessary to release heparin. Miyama (Technological University of Nagaoka): In the case of ionically bound heparin, the release rate of heparin is very critical in preventing thrombus formation: If it is too fast or too slow, the heparinized system is ineffective.