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By Stephen D. Wiviott

* attracts from services of leaders in antiplatelet treatment* Easy-to-use format permits swift shopping* Edited by means of a member of the celebrated TIMI learn workforce with long-standing event within the box

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Inhibition of platelet TXA2 synthesis by traditional NSAIDs is intermittent and incomplete, which contrasts with the irreversible and permanent inhibition of platelet TXA2 synthesis by ASA. 3) [17], which is reflected in their side-effect profiles. NSAIDs with greater COX-2 inhibitory action have fewer GI and renal side effects than NSAIDs that primarily inhibit COX-1. Adverse effects of non-selective NSAIDs The most important adverse effects of NSAIDs result from inhibition of COX in the GI mucosa and kidneys.

Synthesis and use of iodinated nonsteroidal anti-inflammatory drug analogues as crystallographic probes of the prostaglandin H2 synthase cyclooxygenase active site. Biochemistry 1996; 35: 7330–7340. 13 Botting RM. Inhibitors of cyclooxygenases:mechanisms, selectivity and uses. J Physiol Pharmacol 2006; 57 (Supp. 5): 113–124. 14 Vane JR, Botting RM. The mechanism of action of ASA. Thromb Res 2003; 110: 255–258. 15 Fitzgerald G, Patrono C. The coxibs, selective inhibitors of cycooxygenase-2. New Eng J Med 2001; 345: 433–442.

JAMA 2007; 297: 2018–2024. 30 Patrono C, Coller B, Dalen JE et al. Platelet-active drugs: the relationships among dose, effectiveness and side effects. ): 39S–63S. 31 Laine L. Review article: gastrointestinal bleeding with low-dose ASA-what’s the risk? Aliment Pharm Therap 2006; 24: 896–908. 32 Stevenson DD, Szczeklik A. Clinical and pathologic perspectives on ASA sensitivity and asthma. Curr Rev Allergy Clin Immunol 2006; 118: 773–786. 33 Warner TD, Mitchell JA. Cyclooxygenases: new forms, new inhibitors, and lessons from the clinic FASEB J 2004; 18: 790–804.

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